Figure 4From: Hydrogen sulfide provides cardioprotection against myocardial/ischemia reperfusion injury in the diabetic state through the activation of the RISK pathwayNa 2 S therapy increases the expression of Bcl-xL and inhibits Bad following MI/R. (A) Representative immunoblots and densitometric analysis of the expression of (B) Bcl-xL, (C) phosphorylated Bad at Serine residue 112, and (D) total Bad. Experiments were conducted with heart homogenates collected from Sham, Vehicle, and Na2S-treated mice following 30 minutes of ischemia and 4 hours of reperfusion. Values are mean ± SEM. *p < 0.05 vs. Sham.Back to article page